2010 Available at Kornmehl H, Gorouhi F, Konia T, Fung MA, Tartar DM. Red man syndrome is a response or hypersensitive reaction to the antibiotic vancomycin. Vasculitic reaction on the legs. Nonscarring inflammatory alopecia associated with the epidermal growth factor receptor inhibitor gefitinib.

The term was coined in a 1996 report in an attempt to simplify terminology for a syndrome recognized as early as 1959. Cutaneous reactions to recombinant cytokine therapy. DRESS (ie, drug reaction with eosinophilia and systemic symptoms) syndrome or DIHS drug-induced hypersensitivity syndrome.

Iannini P, Mandell L, Felmingham J, Patou G, Tillotson GS. Paquet P, Pierard GE, Quatresooz P. Novel treatments for drug-induced toxic epidermal necrolysis (Lyell's syndrome).

DRESS can progress to a serious condition, especially in patients who have hepatitis. Roujeau JC, Kelly JP, Naldi L, et al. Male-pattern diffuse hair loss. Severe serum sickness reaction to oral and intramuscular penicillin. Heidary N, Naik H, Burgin S. Chemotherapeutic agents and the skin: An update. Association between beta-blockers, other antihypertensive drugs and psoriasis: population-based case-control study. Superficial and mid-dermal perivascular infiltrate of lymphocytes and eosinophils. Antonov D, Kazandjieva J, Etugov D, Gospodinov D, Tsankov N. Drug-induced lupus erythematosus. Confluent necrosis of the epidermis in toxic epidermal necrolysis. Available at Walsh S, Diaz-Cano S, Higgins E, Morris-Jones R, Bashir S, Bernal W, et al. Patients with EM minor recover fully, but relapses are common. Lymphadenopathy is often noted, and hepatosplenomegaly, leukocytosis, eosinophilia, and anemia may be present.Lesions recur in the same area when the offending drug is given (see the image below).

Toxic epidermal necrolysis. These are characterized by the triad of fever, skin eruption, and internal organ involvement, and they usually are associated with intake of anticonvulsant drugs. Cutaneous Reactions to Targeted Therapy. Consider alternative etiologies, especially viral exanthems and bacterial infections. Brauchli YB, Jick SS, Curtin F, Meier CR. Circular, violaceous, edematous plaques that resolve with macular hyperpigmentation is characteristic. Medline.

Mukasa Y, Craven N. Management of toxic epidermal necrolysis and related syndromes.

Most cases are caused by drugs (primarily antibiotics) often in the first few days of administration.

Camilleri M, Pace JL. Patients who react to phenytoin, carbamazepine, and barbiturates will often cross-react to other aromatic anticonvulsants. Use of intravenous immunoglobulin in toxic epidermal necrolysis and Stevens-Johnson syndrome: our current understanding.

Hazin R, Ibrahimi OA, Hazin MI, Kimyai-Asadi A. Stevens-Johnson syndrome: pathogenesis, diagnosis, and management. French LE, Trent JT, Kerdel FA.

Shipley D, Ormerod AD. Note sharp cutoff where clothing blocked exposure.

Cutaneous signs typically begin with erythema on the sides of the fingers, hands, and toes and progress to a widespread eruption (most often morbilliform or urticarial). Yang J, Yang X, Li M. Peripheral blood eosinophil counts predict the prognosis of drug eruptions.
Wu PA, Balagula Y, Lacouture ME, Anadkat MJ. Clindamycin hydrochloride is the hydrated hydrochloride salt of clindamycin.

Clindamycin hydrochloride capsules, USP contain clindamycin hydrochloride, USP equivalent to 150 mg or 300 mg of clindamycin. 14,15 Reactivation of herpes viruses, especially of herpes virus 6, occurs frequently in this syndrome. Generalized fixed drug eruption to piperacillin/tazobactam and review of literature.

Thiol-induced pemphigus tends to resemble pemphigus foliaceus or pemphigus erythematosus; nonthiol eruptions may resemble pemphigus vulgaris or pemphigus vegetans. Lesions are also identical to drug-induced subacute cutaneous lupus erythematosus (SCLE), which is characterized by annular, psoriasiform, nonscarring lesions in a photodistributed pattern.This is the most common pattern of drug eruptions; it is the quintessential drug rash. It typically develops within 2 weeks after the onset of therapy.While largely a drug-induced condition, it can also occur with use of tanning beds and hemodialysis. Target lesions of erythema multiforme. Sweet syndrome often occurs in association with cancers, inflammatory disorders, pregnancy, and medication use.This usually occurs as small wheals that may coalesce or may have cyclical or gyrate forms. It is a reactive process often secondary to infection, but it may be due to medications, especially oral contraceptives and sulfonamides.This is widespread inflammation of the skin (see the image below), and it may result from an underlying skin condition, drug eruption, internal malignancy, or immunodeficiency syndrome.

WHAT IS DRESShttps://www.dresssyndrome.org/what-is-dressQUICK FACTShttps://www.dresssyndrome.org/quick-facts-on-dressWHO WE AREhttps://www.dresssyndrome.org/wh

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